Histomorphometric alteration and cell-type specific modulation of arylhydrocarbon receptor and estrogen receptor expression by 2,3,7,8-tetrachlorodibenzo-p-dioxin and 17β-estradiol in mouse experimental model of endometriosis
This study investigated how TCDD and estradiol alter histomorphometry and aryl hydrocarbon receptor/estrogen receptor expression in specific cell types within a mouse endometriosis model.
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Cited by (6)
- Pathogenesis of Endometriosis: Role of Macrophages in Endometriosis 2022
- Associations between Exposure to Organochlorine Chemicals and Endometriosis: A Systematic Review of Experimental Studies and Integration of Epidemiological Evidence 2021
- Role of Environmental Pollutants in Endometriosis 2015
- Estrogen-independent actions of environmentally relevant AhR-agonists in human endometrial epithelial cells 2010
- Changes in tissue inflammation, angiogenesis and apoptosis in endometriosis, adenomyosis and uterine myoma after GnRH agonist therapy 2009
- REVIEW ARTICLE: Immunopathogenesis of Pelvic Endometriosis: Role of Hepatocyte Growth Factor, Macrophages and Ovarian Steroids 2008
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