Expression of E26 transformation specific (ETS-1) related to angiogenesis in ovarian endometriosis

Sakaguchi H, Hideki Sakaguchi, Jiro Fujimoto, Fujimoto J, Aoki I, Ikumi Aoki, Hiroshi Toyoki, Toyoki H, Eriko Sato, Sato E, Tamaya T, Teruhiko Tamaya
Fertility and sterility · 2004 · vol. 82(2) , pp. 507–510 · doi:10.1016/j.fertnstert.2004.02.103 · PMID:15302318 · W2109863674
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⚙ AI-generated summary by claude@2026-06, 2026-06-10 ⓘ

This study investigated the relationship between ETS-1 expression and angiogenesis in ovarian endometriosis.

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Abstract

ETS-1 in ovarian endometriomas was significantly positively correlated with microvessel counts (MVCs), but ETS-1 and MVC were not significantly altered during the menstrual cycle. Because ETS-1 persistently expresses in the subepithelial area of endometriotic endometrium, this might contribute to the growth of ovarian endometriomas via subepithelial angiogenesis independently of the menstrual cycle.

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Condition tags

endometriosis

MeSH descriptors

Endometriosis Gene Expression Regulation Neovascularization, Pathologic Ovarian Diseases Proto-Oncogene Proteins Transcription Factors Adult Endometriosis Endometriosis Endometriosis Factor VIII Factor VIII Female Gene Expression Regulation Humans Immunohistochemistry Microcirculation Microcirculation Neovascularization, Pathologic Ovarian Diseases

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (31)

Cited by (3)

Source provenance

europepmc
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