Hypoxia-induced autophagy, epithelial to mesenchymal transition, and invasion in the pathophysiology of endometriosis: a perspective
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This paper reviews the role of hypoxia-induced autophagy, epithelial to mesenchymal transition, and invasion in the pathophysiology of endometriosis.
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References (13)
- Altered expression of microRNA-451 in eutopic endometrium of baboons ( Papio anubis ) with endometriosis via openalex
- Autophagy contributes to hypoxia-induced epithelial to mesenchymal transition of endometrial epithelial cells in endometriosis via openalex
- Early Endometriosis in Females Is Directed by Immune-Mediated Estrogen Receptor α and IL-6 Cross-Talk via openalex
- ERβ- and prostaglandin E2-regulated pathways integrate cell proliferation via Ras-like and estrogen-regulated growth inhibitor in endometriosis via openalex
- Estrogen Receptor β Modulates Apoptosis Complexes and the Inflammasome to Drive the Pathogenesis of Endometriosis via openalex
- Hypoxia-inducible factor-1α promotes endometrial stromal cells migration and invasion by upregulating autophagy in endometriosis via openalex
- Serum microRNAs as diagnostic markers of endometriosis: a comprehensive array-based analysis via openalex
- Serum miR-451a Levels Are Significantly Elevated in Women With Endometriosis and Recapitulated in Baboons ( Papio anubis) With Experimentally-Induced Disease via openalex
- W6844153672 via openalex
- W2508914764 via openalex
- W2765750028 via openalex
- W2794241440 via openalex
- W1618972566 via openalex
Cited by (2)
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- europepmc
- last seen: 2026-10-13T06:10:10.580696+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-10-08T21:22:24.294217+00:00
- unpaywall
- last seen: 2026-10-13T06:25:51.201866+00:00
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