Ultrasound study of the myoendometria/border

Angelo Tocci, P. Royo, J. L. Alcázar, M. García-Manero
In: Revista de Medicina de la Universidad de Navarra · 2016 · pp. 3–5 · doi:10.15581/021.53.6155 · W4313163917
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⚙ AI-generated summary by gemini-2.5-flash-lite, 2026-06-08 ⓘ

This study hypothesizes a distinct condition, "ESEMy Unit disruption disease," characterized by thickened myoendometrial borders identified via ultrasound, impacting fertility independently of adenomyosis.

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Abstract

Abnormal thickening of the Endometrial Subendometrial Myometrium Unit (ESEMy Unit, including basal endometrium and inner myometrium) has been detected on imaging and referred to as "diffuse adenomyosis" in infertile patients with proven endometriosis. However, no robust relationship exists between enlargement of the ESEMy Unit and adenomyosis proven on hysterectomy specimen examination; moreover, if any correlation exists, it lacks histological validation in women wishing to preserve fertility. While adenomyosis effects on fertility, if any, remain elusive, thickening of the ESEMy Unit have been consistently linked to fertility impairment in both experimental and clinical models. The hypothesis tested herein is that a novel condition exists, called "ESEMy Unit disruption disease"; it is idemiologically different from adenomyosis, diagnosable on imaging and bears a clear impact on human fertility through various mechanisms. A new wave of good quality studies may be elicited by a clear distinction between adenomyosis and the "ESEMy Unit disruption disease".

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Condition tags

endometriosisadenomyosis

Citation neighborhood (2-hop)

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. Outer rings show 2-hop neighbours — papers reached through the immediate citers/citees. [ collapse to 1-hop ]

References (16)

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