Carla de Azevedo Piccinato

No ORCID on file · 18 papers in corpus · active 2012-2022

Study types

  • article 10
  • review 5
  • dataset 1
  • erratum 1
  • other 1

Condition tags

  • endometriosis 17
  • mesh:D004715 12
  • infertility 3
  • die_deep_infiltrating 2
  • chronic_pelvic_pain 1
  • mesh:D017699 1
article 2022
International journal of molecular sciences ·doi:10.3390/ijms23052476

Endometriosis causes immunological and cellular alterations. Endometriosis lesions have lower levels of lamin b1 than the endometrium. Moreover, high levels of pro-inflammatory markers are observed in the peritoneal fluid, follicular fluid,…

other 2022
Frontiers in molecular biosciences ·doi:10.3389/fmolb.2022.854991

Endometriosis is a highly prevalent gynecological disease characterized by lesions in different sites. Regulation of specific estrogen pathways may favor the formation of distinct microenvironments and the progression of endometriosis. Howe…

article 2022
Einstein (Sao Paulo, Brazil) ·doi:10.31744/einstein_journal/2022ao0086

OBJECTIVE: To evaluate the prevalence of sonographic signs suggestive of deep infiltrative endometriosis and endometriomas in patients referred for transvaginal sonography as part of a routine annual gynecological evaluation. We also descri…

dataset 2021
·doi:10.6084/m9.figshare.14317095

Abstract Objective The present study is a systematic review of the literature to assess whether the presence of endometriosis determines or contributes to adverse obstetric outcomes. Data Sources The present work was carried out at the Hosp…

review 2020
Journal of translational medicine ·doi:10.1186/s12967-020-02471-0

Endometriosis is a chronic inflammatory hormone-dependent condition associated with pelvic pain and infertility, characterized by the growth of ectopic endometrium outside the uterus. Given its still unknown etiology, treatments usually aim…

article 2020
·doi:10.1016/j.fertnstert.2020.08.243
review 2020
Revista brasileira de ginecologia e obstetricia : revista da Federacao Brasileira das Sociedades de Ginecologia e Obstetricia ·doi:10.1055/s-0040-1708885

Abstract Objective The present study is a systematic review of the literature to assess whether the presence of endometriosis determines or contributes to adverse obstetric outcomes. Data Sources The present work was carried out at the Hosp…

review 2019
Reproduction (Cambridge, England) ·doi:10.1530/rep-18-0618

The objective is to study the significance of altered interleukin levels in endometriosis-related infertility or pelvic pain. The present systematic review and meta-analysis includes a discussion on the roles of interleukin in the physiopat…

article 2019
Medicina (Kaunas, Lithuania) ·doi:10.3390/medicina55090520

Background and Objectives: The presence of endometrial-like tissue outside the uterine cavity is a key feature of endometriosis. Although endometriotic lesions appear to be histologically quite similar to the eutopic endometrium, detailed s…

article 2018
article 2018
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1177/1933719118764255
review 2018
Journal of molecular endocrinology ·doi:10.1530/jme-17-0297

Endometriosis is an incurable hormone-dependent inflammatory disease that causes chronic pelvic pain and infertility characterized by implantation and growth of endometrial tissue outside the uterine cavity. Symptoms have a major impact on …

erratum 2017
Journal of reproductive immunology ·doi:10.1016/j.jri.2017.05.004
review 2017
Journal of reproductive immunology ·doi:10.1016/j.jri.2017.04.003
article 2016
Reproduction (Cambridge, England) ·doi:10.1530/rep-15-0581

Endometriosis is an estrogen-dependent disease affecting up to 10% of all premenopausal women. There is evidence that different endometriosis sites show distinct local estrogen concentration, which, in turn, might be due to a unique local e…

article 2016
Molecular and cellular biochemistry ·doi:10.1007/s11010-016-2899-3
article 2015
The Journal of steroid biochemistry and molecular biology ·doi:10.1016/j.jsbmb.2015.12.025
article 2012
·doi:10.1016/j.fertnstert.2012.07.778