Host immunity and KLF 11 deficiency together promote fibrosis in a mouse model of endometriosis.
article
OA: closed
CC0
⤵ 3 in-corpus citations
Limited metadata. Only one source feed has indexed
this record so far — no abstract, full text, or open-access copy is
available through Endo Lab. The
publisher's page (linked below)
is the canonical location for the actual content. If you have institutional
access, use "Find at my library".
AI-generated summary
This study investigated how host immunity and KLF 11 deficiency interact to drive fibrosis in a mouse model of endometriosis.
One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works
My notes (saved in your browser only)
Condition tags
MeSH descriptors
Citation neighborhood
Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.
References (53)
- A Novel Role of the Sp/KLF Transcription Factor KLF11 in Arresting Progression of Endometriosis via openalex
- Cancer-Associated Mutations in Endometriosis without Cancer via openalex
- Cellular, Histologic, and Molecular Changes Associated With Endometriosis and Ovarian Cancer via openalex
- Deeply infiltrating endometriosis affecting the rectum and lymph nodes via openalex
- Endometriosis allergic or autoimmune disease: pathogenetic aspects - a case control study via openalex
- EPIDEMIOLOGY OF ENDOMETRIOSIS via openalex
- Epigenetic Modulation of Collagen 1A1: Therapeutic Implications in Fibrosis and Endometriosis1 via openalex
- Epigenetic regulation and T-cell responses in endometriosis – something other than autoimmunity via openalex
- Epigenetic Therapy: Novel Translational Implications for Arrest of Environmental Dioxin-Induced Disease in Females via openalex
- Experimental Endometriosis via openalex
- Ignored Because It Is Benign – It Is Time to Treat Endometriosis as if It Were Cancer via openalex
- Immunology of endometriosis via openalex
- Immunopathogenesis of endometriosis – a novel look at an old problem via openalex
- Induction of endometriosis in mice: A new model sensitive to estrogen via openalex
- KRAS Activation and over-expression of SIRT1/BCL6 Contributes to the Pathogenesis of Endometriosis and Progesterone Resistance via openalex
- Recent insights on the genetics and epigenetics of endometriosis via openalex
- The endometria of patients with endometriosis show higher expression of class I human leukocyte antigen than the endometria of healthy women via openalex
- The histopathological features of the surgical endometriosis model using systemic autoimmune disease-prone mice via openalex
- T helper 17 axis and endometrial macrophage disruption in menstrual effluent provides potential insights into the pathogenesis of endometriosis via openalex
- Theories on the Pathogenesis of Endometriosis via openalex
- The Wnt/β-catenin signaling in endometriosis, the expression of total and active forms of β-catenin, total and inactive forms of glycogen synthase kinase-3β, WNT7a and DICKKOPF-1 via openalex
- Treatment with Bazedoxifene, a Selective Estrogen Receptor Modulator, Causes Regression of Endometriosis in a Mouse Model via openalex
- W3009038333 via openalex
- W3190429688 via openalex
- W3208517554 via openalex
- W4211081176 via openalex
- W4221030787 via openalex
- W4249777411 via openalex
- W6604609371 via openalex
- W6683826416 via openalex
- W6811038925 via openalex
- W2264712737 via openalex
- W1965676298 via openalex
- W1972193141 via openalex
- W1985929046 via openalex
- W2058351795 via openalex
- W2062426992 via openalex
- W2067407021 via openalex
- W2096790979 via openalex
- W2097065948 via openalex
- W2114104545 via openalex
- W2116591759 via openalex
- W2117943066 via openalex
- W2134513968 via openalex
- W2137586531 via openalex
- W2143417145 via openalex
- W2163647575 via openalex
- W2171195379 via openalex
- W114318986 via openalex
- W2490109341 via openalex
- W2772343293 via openalex
- W2884952568 via openalex
- W2965589450 via openalex
Cited by (3)
Source provenance
- europepmc
- last seen: 2026-06-12T06:13:51.797165+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-06-01T00:33:40.069348+00:00
License: CC0
· commercial use OK