Pathogenesis

review OA: closed CC0 ⤵ 23 in-corpus citations
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AI-generated summary by claude@2026-06, 2026-06-07

This review examines historical theories on the histogenesis of endometriotic lesions and current evidence implicating immune system components in the disease's pathophysiology.

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AI-generated deep summary by claude@2026-06, 2026-06-07

This 1997/2008 review paper examines competing hypotheses for the pathogenesis of endometriosis, noting that no single theory accounts for all cases and that multiple mechanisms likely contribute. It first reviews evidence on different aspects of the histogenesis of endometriotic lesions and then highlights newer findings implicating components of the immune system in disease development, including macrophages and natural killer cells, as well as cytokine-related pathways. A key limitation is that the discussion is a synthesis of prior studies rather than new experimental data, and it does not establish a unified mechanism. This paper is centrally about endometriosis — it reviews histogenesis theories and immune-system factors (macrophages, natural killer cells, cytokines) implicated in its pathogenesis.

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Abstract

There are many hypotheses concerning the pathogenesis of endometriosis, though no single theory can explain all cases. It is likely that several mechanisms are involved. Early studies concentrated on the histogenesis of the endometriotic lesion. Recent evidence has implicated components of the immune system in the pathogenesis of endometriosis. This review considers the evidence for different theories of the histogenesis of endometriosis and discusses possible immune factors that may be involved in the pathophysiology of the disease.

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Condition tags

endometriosis

MeSH descriptors

Endometriosis Endometrium Immunity, Cellular Peritoneal Diseases Ascitic Fluid Ascitic Fluid Cytokines Cytokines Endometriosis Endometriosis Endometriosis Endometrium Female Humans Immunity, Cellular Killer Cells, Natural Killer Cells, Natural Leukocytes Leukocytes Macrophages

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

Cited by (23)

Source provenance

europepmc
last seen: 2026-06-11T06:19:48.454388+00:00
openalex
last seen: 2026-06-10T17:14:06.276822+00:00
pubmed
last seen: 2026-05-13T22:10:52.568893+00:00
License: CC0 · commercial use OK