What somatic-lineage analysis can - and cannot - show about the cellular ancestry of endometriosis lesions

Citation neighborhood (2-hop)

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. Outer rings show 2-hop neighbours — papers reached through the immediate citers/citees. [ collapse to 1-hop ]

References (15)

• Cancer driver mutations in endometriosis: Variations on the major theme of fibrogenesis via openalex
• Clonal Expansion and Diversification of Cancer-Associated Mutations in Endometriosis and Normal Endometrium via openalex
• Endometriosis caused by retrograde menstruation: now demonstrated by DNA evidence via openalex
• Epigenetic age provides insight into tissue origin in endometriosis via openalex
• Identification of displaced endometrial glands and embryonic duct remnants in female fetal reproductive tract: possible pathogenetic role in endometriotic and pelvic neoplastic processes via openalex
• Independent development of endometrial epithelium and stroma within the same endometriosis via openalex
• Molecular analysis suggests oligoclonality and metastasis of endometriosis lesions across anatomically defined subtypes via openalex
• Müllerianosis: four developmental (embryonic) mullerian diseases via openalex
• Mutation and methylation profiles of ectopic and eutopic endometrial tissues via openalex
• Peritoneal endometriosis due to the menstrual dissemination of endometrial tissue into the peritoneal cavity via openalex
• Variation in endometriosis aetiology, potential impact on lesion heterogeneity and the consequences for research and treatment via openalex
• W2883043301 via openalex
• W3020114628 via openalex
• W2015182193 via openalex
• W7130510496 via openalex

Source provenance

openalex

last seen: 2026-10-15T06:00:39.803327+00:00