Correction: Pathophysiology of Endometriosis: Role of High Mobility Group Box-1 and Toll-Like Receptor 4 Developing Inflammation in Endometrium
This paper corrects the DOI for an article investigating the role of High Mobility Group Box-1 and Toll-Like Receptor 4 in developing endometrial inflammation in endometriosis.
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This article is a correction to a previously published PLoS ONE paper on endometriosis pathophysiology, specifically amending an error in the penultimate sentence under the “Participants” subheading in the Materials and Methods section. The corrected statement specifies that the study was approved by the institutional review board of Severance Hospital, Yonsei University College of Medicine (4-2012-0458). No additional experimental data, results, or scientific conclusions from the original research are changed in this correction. This paper is centrally about endometriosis — it is explicitly a correction associated with a study on HMGB1 and Toll-like receptor 4–linked inflammation in the endometrium.
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Too few in-corpus citations on either side for a chart; here are the lists.
Cites (1)
Cited by (4)
- Atherosclerotic cardiovascular disease in women with endometriosis: a systematic review of risk factors and prospects for early surveillance 2019
- HMGB1 Mediated Inflammation and Autophagy Contribute to Endometriosis 2021
- Association of Inflammatory Markers/Cytokines with Cardiovascular Risk Manifestation in Patients with Endometriosis 2021
- Endometriosis promotes atherosclerosis in a murine model 2022
References (1)
Cited by (4)
- Endometriosis promotes atherosclerosis in a murine model 2022
- HMGB1 Mediated Inflammation and Autophagy Contribute to Endometriosis 2021
- Association of Inflammatory Markers/Cytokines with Cardiovascular Risk Manifestation in Patients with Endometriosis 2021
- Atherosclerotic cardiovascular disease in women with endometriosis: a systematic review of risk factors and prospects for early surveillance 2019
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