Endometriosis

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This book clarifies endometriosis pathogenesis and pathophysiology with contributions from 40 experts covering stem cells, inflammation, and epigenetics, alongside practical prevention and treatment strategies.

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AI-generated deep summary by claude@2026-06, 2026-06-07 · read from full text

This 2014 Springer book entitled “Endometriosis: Pathogenesis and Treatment” examines the pathogenesis and pathophysiology of endometriosis and synthesizes related therapeutic approaches for clinicians and researchers. It covers multiple mechanistic areas, including immune components (NK cells, macrophages), inflammation and cytokines, epigenetics, prostaglandin and sex steroid pathways, aromatase, apoptosis, nerve fibers, and malignant transformation, with a second section describing recent tools for prevention, imaging, biomarkers, medical and surgical treatments, recurrence prevention, and infertility treatments. The key finding across the book is an integrated, multidisciplinary framework linking diverse biological processes to disease behavior and management options. The main limitation explicitly noted is none beyond the general scope description of this clinician-oriented, expert-authored reference text. This paper is centrally about endometriosis — it is a comprehensive book on endometriosis pathogenesis and treatment.

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Overview - Helps clarify pathogenesis and pathophysiology of endometriosis - Covers all essential information for clinicians, including prevention and practical treatment - Written by nearly 40 internationally renowned researchers Access this book Tax calculation will be finalised at checkout Other ways to access About this book Endometriosis is one of the most common gynecological diseases, causing pelvic pain, infertility, and cancer. The condition reduces the quality of life for the estimated 10% of menstruating women who are affected by it. This book provides a clear insight into the pathogenesis and pathophysiology of endometriosis—essential information for clinicians intending to successfully treat the condition. The first part covers a range of topics including stem cells, NK cells, macrophages, inflammation, cytokines, epigenetics, prostaglandin, sex steroids, aromatase, apoptosis, nerve fibers, and malignant transformation to aid in understanding the pathogenesis and pathophysiology of the disease. The second part provides useful tools for therapy, specifically, notable recent techniques in prevention, imaging, biomarkers, medical treatments, surgical treatments, prevention of recurrence, and infertility treatments. Each chapter was written by an expert in the field, with nearly 40 researchers and clinicians contributing to the work. This book will be highly useful to clinicians as well as to research scientists who are interested in this enigmatic disease. Similar content being viewed by others Table of contents (29 chapters) - Introduction - Basic Science Editors and Affiliations About the editor Accessibility Information Accessibility information for this book is coming soon. We're working to make it available as quickly as possible. Thank you for your patience. Bibliographic Information Book Title: Endometriosis Book Subtitle: Pathogenesis and Treatment Editors: Tasuku Harada DOI: https://doi.org/10.1007/978-4-431-54421-0 Publisher: Springer Tokyo eBook Packages: Medicine, Medicine (R0) Copyright Information: Springer Japan 2014 Hardcover ISBN: 978-4-431-54420-3Published: 20 August 2014 Softcover ISBN: 978-4-431-56157-6Published: 17 September 2016 eBook ISBN: 978-4-431-54421-0Published: 06 August 2014 Edition Number: 1 Number of Pages: IX, 477 Number of Illustrations: 25 b/w illustrations, 44 illustrations in colour Topics: Gynecology, Urology, Obstetrics/Perinatology/Midwifery

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endometriosis

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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

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