Hormonresistenz in der medikamentösen Therapie der Endometriose

Q. Le, Sebastian Franik, Ludwig Kiesel, Sebastian Daniel Schäfer
In: Gynäkologische Endokrinologie · 2020 · vol. 18(4) , pp. 252–257 · doi:10.1007/s10304-020-00346-7 · W3091006938
article OA: closed CC0
Limited metadata. Only one source feed has indexed this record so far — no abstract, full text, or open-access copy is available through Endo Lab. The publisher's page (linked below) is the canonical location for the actual content. If you have institutional access, use "Find at my library".
View at publisher → View on OpenAlex
⚙ AI-generated summary by claude@2026-06+body, 2026-06-07 ⓘ

This review discusses progesterone resistance in endometriosis, its origins, consequences, and interventions, citing studies on various hormonal therapies.

One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works

⚙ AI-generated deep summary by claude@2026-06, 2026-06-07 ⓘ

This review article examines hormone resistance as a mechanism underlying variable efficacy of medical therapy in endometriosis, focusing on progesterone resistance and related estrogen/progesterone signaling pathways. It synthesizes evidence from the literature describing origins of progesterone resistance, consequences for treatment response, and proposed interventions, including how progesterone receptor status may predict response to progestins and why signaling may fail. A major limitation is that, as a narrative literature synthesis, it does not present new patient data or a uniform systematic method for comparing studies. Relevance to endometriosis: the paper’s central theme is hormone resistance in the pharmacologic treatment of endometriosis, specifically mechanisms such as progesterone signaling dysfunction and therapeutic success versus failure.

Read from the paper's body, not the abstract. Not a substitute for reading the paper. No clinical advice. How this works

My notes (saved in your browser only)

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (20)

Source provenance

openalex
last seen: 2026-06-10T17:14:06.276822+00:00
unpaywall
last seen: 2026-06-02T02:00:03.124865+00:00
License: CC0 · commercial use OK