Qi S

No ORCID on file · 8 papers in corpus · active 2015-2025

Study types

  • article 2
  • other 1

Condition tags

  • endometriosis 2
  • infertility 1
  • adenomyosis 1
2025
Progestin-primed ovarian stimulation protocol in patients undergoing assisted reproductive technology.
Frontiers in reproductive health ·doi:10.3389/frph.2025.1719930

PurposeTo evaluate the effectiveness and safety of the novel protocol-progestin-primed ovarian stimulation (PPOS) protocol during controlled ovarian hyperstimulation (COH), in patients undergoing in vitro fertilization/intracytopalsmic sper…

2023
Association of chorioamnionitis with infertility treatment and subsequent neonatal outcomes in the US: a population-based cohort study.
BMC pregnancy and childbirth ·doi:10.1186/s12884-023-05619-0

BackgroundChorioamnionitis (CAM) is a common risk factor for preterm births, resulting in several adverse outcomes. The association between infertility treatment and CAM is unclear. Therefore, this study examined the association between inf…

2022
Effect of Coenzyme Q10 and transcutaneous electrical acupoint stimulation in assisted reproductive technology: a retrospective controlled study.
Reproductive biology and endocrinology : RB&E ·doi:10.1186/s12958-022-01043-9

PurposeTo investigate the effects of coenzyme Q10 (CoQ10) and transcutaneous electrical acupoint stimulation (TEAS) pretreatment on pregnancy in patients with poor ovarian response (POR).MethodsA total of 330 POR patients who were pretreate…

other 2022
IGF2BP2 promotes the progression of ovarian endometriosis by regulating m6A-modified MEIS2 and GATA6
The international journal of biochemistry & cell biology ·doi:10.1016/j.biocel.2022.106296

BACKGROUND: m6A-RNA modification mediated by the N6-methyladenosine RNA methylation-related molecule methyltransferase-like 3 has been implicated in the progression of endometriosis. However, the functions of other m6A regulators, especiall…

2020
Structure of the human gonadotropin-releasing hormone receptor GnRH1R reveals an unusual ligand binding mode.
Nature communications ·doi:10.1038/s41467-020-19109-w

Gonadotrophin-releasing hormone (GnRH), also known as luteinizing hormone-releasing hormone, is the main regulator of the reproductive system, acting on gonadotropic cells by binding to the GnRH1 receptor (GnRH1R). The GnRH-GnRH1R system is…

article 2018
Melatonin inhibits 17β-estradiol-induced migration, invasion and epithelial-mesenchymal transition in normal and endometriotic endometrial epithelial cells
Reproductive biology and endocrinology : RB&E ·doi:10.1186/s12958-018-0375-5

BACKGROUND: Melatonin is a potential therapeutic agent for endometriosis, but its molecular mechanism is unclear. Here, we investigated the effect of melatonin on the epithelial-mesenchymal transition (EMT) in endometriotic endometrial epit…

2016
Metformin inhibits 17β-estradiol-induced epithelial-to-mesenchymal transition via βKlotho-related ERK1/2 signaling and AMPKα signaling in endometrial adenocarcinoma cells.
Oncotarget ·doi:10.18632/oncotarget.7040

The potential role of metformin in treating endometrial cancer remains to be explored. The current study investigated the role of metformin in 17β-estradiol-induced epithelial-mesenchymal transition (EMT) in endometrial adenocarcinoma cells…

article 2015
Aberrant expression of Notch1/numb/snail signaling, an epithelial mesenchymal transition related pathway, in adenomyosis
Reproductive biology and endocrinology : RB&E ·doi:10.1186/s12958-015-0084-2

BACKGROUND: Epithelial mesenchymal transition (EMT) is involved in the pathogenesis of adenomyosis, and Notch signaling is crucial to EMT. The objective of this study was to explore Notch1/Numb/Snail signaling in adenomyosis. METHODS: The e…