Michael D. Slater

article 2006

Human growth hormone and interleukin-6 are upregulated in endometriosis and endometrioid adenocarcinoma

Acta histochemica ·doi:10.1016/j.acthis.2006.01.004

In this retrospective and quantitated study on banked tissue we found that, compared to normal uterine epithelial cells, growth hormone (GH) is increased 3.4-fold in endometriosis and 3.8-fold in endometrial adenocarcinoma. Similarly, inter…

article 2005

Adehesion Molecules in Endometriosis and Endometrioid Adenocarcinoma Show Altered Expressions Suggesting a Commonality Between These Diseases

·doi:10.1179/his.2005.28.3.185

(2005). Adehesion Molecules in Endometriosis and Endometrioid Adenocarcinoma Show Altered Expressions Suggesting a Commonality Between These Diseases. Journal of Histotechnology: Vol. 28, No. 3, pp. 185-190.

article 2005

Adehesion Molecules in Endometriosis and Endometrioid Adenocarcinoma Show Altered Expressions Suggesting a Commonality Between These Diseases

·doi:10.1179/014788805794775172

The cadherin family of proteins is significantly deexpressed in endometriosis and even more so in endometrioid adenocarcinoma. A similar finding was observed for the cadherin-associated cell adhesion protein plakoglobin. E-cadherin was not …

article 2005

Endometriotic Cells Exhibit Metaplastic Change and Oxidative DNA Damage as Well as Decreased Function, Compared to Normal Endometrium

Journal of molecular histology ·doi:10.1007/s10735-005-3802-9

A widely accepted theory of the etiology of endometriosis is that it originates from the implantation and invasion of cells from retrograde menstruation to various sites in the body particularly the pelvic peritoneal cavity. Little is known…