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- endometriosis 11
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BACKGROUND: Endometriosis (EMs) is a condition characterized by the growth of endometrial tissue outside the uterine cavity. Although this condition is benign, it has cancer-like features. N6-methyladenosine (m6A) is a common RNA modificati…
Endometriosis is defined as an oestrogen-dependent and inflammatory gynaecological disease of which the pathogenesis remains unclear. This study aimed to investigate the cellular heterogeneity and reveal the effect of CD8+ T cells on the pr…
BACKGROUND: Endometriosis is a benign gynecological disease with the feature of estrogen dependence and inflammation. The function of autophagy and the correlation with inflammation were not yet revealed. METHODS: Autophagosomes were detect…
PROBLEM: The application of primary eutopic endometrial cells from endometriosis patients in research is restricted for short life span, dedifferentiation of hormone responsiveness. METHOD OF STUDY: Human telomerase reverse transcriptase (…
Endometriosis is an estrogen-dependent disease associated with pain and infertility. The objective of this study was to determine the expression of ZEB1 in endometriosis and its role in 17β-estradiol (E2)-induced epithelial-mesenchymal tran…
AIM: Lipoxin A4 (LXA4 ) can function as an endogenous 'breaking signal' in inflammation and plays an important role in the progression of endometriosis. The proteome responses to interleukin-1β (IL-1β) or LXA4 in human endometriotic stromal…
Objective:Epithelial–mesenchymal transition (EMT) is essential for embryogenesis, fibrosis, and tumor metastasis. Aberrant EMT phenomenon has been reported in endometriotic tissues of patients with endometriosis (EM). In this study, we furt…
Objective:Epithelial–mesenchymal transition (EMT) is essential for embryogenesis, fibrosis, and tumor metastasis. Aberrant EMT phenomenon has been reported in endometriotic tissues of patients with endometriosis (EM). In this study, we furt…
Supplementary Material for Lipoxin A4 Suppresses Estrogen-Induced Epithelial–Mesenchymal Transition via ALXR-Dependent Manner in Endometriosis by Rong-Feng Wu, Zhi-Xiong Huang, Jing Ran, Song-Juan Dai, Dian-Chao Lin, Tai-Wei Ng, Qing-Xi Che…
Supplementary Material for Lipoxin A4 Suppresses Estrogen-Induced Epithelial–Mesenchymal Transition via ALXR-Dependent Manner in Endometriosis by Rong-Feng Wu, Zhi-Xiong Huang, Jing Ran, Song-Juan Dai, Dian-Chao Lin, Tai-Wei Ng, Qing-Xi Che…