Rosa I Barañao

No ORCID on file · 6 papers in corpus · active 2002-2019

Study types

  • article 5
  • other 1

Condition tags

  • endometriosis 6
  • infertility 1
article 2019
PI3K/AKT pathway is altered in the endometriosis patient’s endometrium and presents differences according to severity stage
Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology ·doi:10.1080/09513590.2019.1680627

Based on the inflammatory nature and hormone-dependency of endometriosis, PI3K/AKT signaling appears to influence its progression. Could the endometriosis stages be linked to differential changes in PI3K/AKT pathway regulation? The objectiv…

article 2016
Inhibition of Hyaluronic Acid Synthesis Suppresses Angiogenesis in Developing Endometriotic Lesions
PloS one ·doi:10.1371/journal.pone.0152302

BACKGROUND: The development and long-term survival of endometriotic lesions is crucially dependent on an adequate vascularization. Hyaluronic acid (HA) through its receptor CD44 has been described to be involved in the process of angiogenes…

article 2014
Targeting galectin‐1‐induced angiogenesis mitigates the severity of endometriosis
The Journal of Pathology ·doi:10.1002/path.4397

Endometriosis is characterized by the presence of endometrial tissue outside the uterus that causes severe pelvic pain and infertility in women of reproductive age. Although not completely understood, the pathophysiology of the disease invo…

article 2003
Effect of GnRH analogues on apoptosis and release of interleukin-1  and vascular endothelial growth factor in endometrial cell cultures from patients with endometriosis
Human Reproduction ·doi:10.1093/humrep/deg356

BACKGROUND: The aim of the present study was to evaluate the effect of GnRH analogues on the in-vitro eutopic endometrial cell apoptosis and release of interleukin-1beta (IL-1beta) and vascular endothelial growth factor (VEGF). METHODS: Bio…

article 2003
Gonadotropin-releasing hormone agonist induces apoptosis and reduces cell proliferation in eutopic endometrial cultures from women with endometriosis
Fertility and Sterility ·doi:10.1016/s0015-0282(03)00769-6
other 2002
Oral contraceptives suppress cell proliferation and enhance apoptosis of eutopic endometrial tissue from patients with endometriosis
Fertility and Sterility ·doi:10.1016/s0015-0282(02)03099-6

OBJECTIVE: To evaluate the effects of administering combination oral contraceptives (COCs) to patients with endometriosis on the regulation of cell growth in the eutopic endometrium. DESIGN: Prospective study. SETTING: Research institute …