H M Gebel

No ORCID on file · 5 papers in corpus · active 1987-1998

Study types

  • article 4
  • other 1

Condition tags

  • mesh:D004715 5
  • endometriosis 5
  • infertility 1
article 1998
Fertility and Sterility ·doi:10.1016/s0015-0282(98)00073-9
article 1995
American journal of reproductive immunology (New York, N.Y. : 1989) ·doi:10.1111/j.1600-0897.1995.tb00958.x

PROBLEM: Previous studies have established that in vitro proliferation of endometrial cells is enhanced by peripheral blood monocytes (PBM) and suppressed by peritoneal macrophages (PM) from patients with endometriosis but only suppressed b…

article 1994
Acta obstetricia et gynecologica Scandinavica. Supplement

It is well recognized that cell-mediated immune responses contribute to the elimination of foreign antigens and cells from the invading organism. It is also likely that the immune system can recognize and eliminate altered or misplaced auto…

other 1993
American journal of reproductive immunology (New York, N.Y. : 1989) ·doi:10.1111/j.1600-0897.1993.tb00576.x

PROBLEM: The etiology and/or pathogenesis of endometriosis may involve aspects of both humoral and cellular immunity. METHOD: In this investigation, we analyzed the ability of B lymphocytes from distinct patient groups for production of Ig…

article 1987
Obstetrics and gynecology clinics of North America ·doi:10.1159/000414847

Endometriosis appears to be a systemic disease with alterations in both humoral and cell-mediated immunity. An immune defect may allow ectopic implantation of the endometrial cells with subsequent development of autoantibodies.