Guillermo Antiñolo

ORCID: 0000-0002-2113-074X · 3 papers in corpus · active 2003-2005

Study types

  • article 3

Condition tags

  • endometriosis 3
article 2005
Polymorphisms in the promoter regions of FAS and FASL genes as candidate genetic factors conferring susceptibility to endometriosis
International journal of molecular medicine ·doi:10.3892/ijmm.15.5.865

Although the pathophysiological mechanisms leading to endometriosis remain unknown, several hypothesis have been proposed, including a dysregulation of the normal apoptotic process which takes place in the endometrium. One of the apoptotic …

article 2004
Analysis of the involvement of CCR5- 32 and CCR2-V64I variants in the development of endometriosis
Molecular human reproduction ·doi:10.1093/molehr/gah026

Several arguments support the proposal that the cytokine network plays a critical role in the aetiology of endometriosis. Among various chemokines, regulated-on-activation, normal-T-cell-expressed and -secreted (RANTES) and monocyte chemota…

article 2003
Evaluation of germline sequence variants within the promoter region of RANTES gene in a cohort of women with endometriosis from Spain
Molecular human reproduction ·doi:10.1093/molehr/gag057

The RANTES (regulated upon activation normal T cells expressed and secreted) chemokine, is known to be expressed in endometriotic lesions in a concentration correlating with the severity of endometriosis. Since it has been widely demonstrat…