Bai JB

No ORCID on file · 3 papers in corpus · active 2017-2025

Study types

  • other 2
  • article 1

Condition tags

  • endometriosis 3
other 2025
A novel mechanism of FTO modulating the progression of endometriosis through mediating the m6A methylation of GEF-H1 in a YTHDF1-dependent manner
Molecular medicine (Cambridge, Mass.) ·doi:10.1186/s10020-025-01130-8

BACKGROUND: Endometriosis (EMs) is a condition characterized by the growth of endometrial tissue outside the uterine cavity. Although this condition is benign, it has cancer-like features. N6-methyladenosine (m6A) is a common RNA modificati…

article 2022
Lipoxin A4 depresses inflammation and promotes autophagy via AhR/mTOR/AKT pathway to suppress endometriosis
American journal of reproductive immunology (New York, N.Y. : 1989) ·doi:10.1111/aji.13659

BACKGROUND: Endometriosis is a benign gynecological disease with the feature of estrogen dependence and inflammation. The function of autophagy and the correlation with inflammation were not yet revealed. METHODS: Autophagosomes were detec…

other 2017
Effect of interleukin-1β and lipoxin A4 in human endometriotic stromal cells: Proteomic analysis
Journal of Obstetrics and Gynaecology Research ·doi:10.1111/jog.13201

AIM: Lipoxin A4 (LXA4 ) can function as an endogenous 'breaking signal' in inflammation and plays an important role in the progression of endometriosis. The proteome responses to interleukin-1β (IL-1β) or LXA4 in human endometriotic stromal…