Matthew Dyson

No ORCID on file · 5 papers in corpus
article 2017
Reproduction (Cambridge, England) ·doi:10.1530/rep-16-0592

Decidualization alters multiple molecular pathways in endometrium to permit successful embryo implantation. We have reported that paracrine factors, including retinoids, secreted from progesterone-treated endometrial stromal cells, act on n…

article 2016
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1177/1933719116632920

ObjectiveFenretinide is a synthetic retinoid analogue that promotes apoptosis but has decreased toxicity when compared to other retinoids. We have previously shown that retinoic acid (RA) production in endometriotic tissue is decreased, res…

review 2015
Seminars in reproductive medicine ·doi:10.1055/s-0035-1554053

Endometriosis has been initially described as the presence of ectopic endometrial tissue on pelvic organs or in extrapelvic sites; and this has been used as its key pathologic feature ever since. Endometriosis responds to fluctuations in es…

review 2012
Seminars in reproductive medicine ·doi:10.1055/s-0031-1299596

Endometriosis is an estrogen-dependent disease. The biologically active estrogen, estradiol, aggravates the pathological processes (e.g., inflammation and growth) and the symptoms (e.g., pain) associated with endometriosis. Abundant quantit…

article 2011
Human Reproduction ·doi:10.1093/humrep/der172

BACKGROUND: Retinoic acid (RA) regulates key biological processes, including differentiation, apoptosis and cell survival. RA mediates induction of 17 beta-hydroxysteroid dehydrogenase type 2 mRNA, catalyzing the conversion of estradiol to …