Prentice A

No ORCID on file · 33 papers in corpus
observational 1996
Human Reproduction ·doi:10.1093/oxfordjournals.humrep.a019023

Active endometriosis is characterized by hypervascularization both within and surrounding the implant; therefore the presence of angiogenic factors in the peritoneal environment would be of great importance. Vascular endothelial growth fact…

other 1994
British journal of obstetrics and gynaecology ·doi:10.1111/j.1471-0528.1994.tb13187.x

OBJECTIVE: To compare the expression of cell adhesion molecules by endometrium and endometriosis. DESIGN: A comparative study of integrin expression, determined immunohistochemically, in eutopic and ectopic endometrium biopsied synchronous…

other 1993
BMJ (Clinical research ed.) ·doi:10.1136/bmj.306.6877.585-d

proportions of abnormal findings.Although many negative results will be clinically useful, an unduly high negative percentage would suggest that the referral threshold needed to be reviewed.

other 1993
Human Reproduction ·doi:10.1093/oxfordjournals.humrep.a137856

Growth factors play a role in the cyclical growth and vascularization of normal endometrium. Abnormal endometrial proliferation and neovascularization may result in endometriosis. This study determines the presence and localization of acidi…

observational 1992
British journal of obstetrics and gynaecology ·doi:10.1111/j.1471-0528.1992.tb13756.x

OBJECTIVE: To study epidermal growth factor (EGF) receptor expression in endometrium throughout the menstrual cycle and to compare EGF-receptor expression in endometrium from patients with endometriosis with receptor expression in synchrono…

observational 1992
Human Reproduction ·doi:10.1093/oxfordjournals.humrep.a137848

Endometriosis is an oestrogen dependent condition and it is expected that the tissue of origin of endometriosis will express receptors for the ovarian steroids. Two epithelia, endometrium and peritoneal mesothelium, are the potential parent…

1991
lab-animal 1991
Human Reproduction ·doi:10.1093/oxfordjournals.humrep.a137512

Endometrial proliferation is stimulated by oestradiol. The precise mechanism is poorly understood, but may be mediated by epidermal growth factor (EGF). The aim of the present study was to assess the effects of oestradiol and EGF on glandul…