17β-Estradiol and lipopolysacchride additively promote pelvic inflammation and growth of endometriosis
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This study investigated the combined effects of 17β-estradiol and lipopolysaccharide, finding they additively promote pelvic inflammation and the growth of endometriosis.
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Cited by (4)
- Endometriosis‐associated infertility: aspects of pathophysiological mechanisms and treatment options 2016
- Enhanced epithelial to mesenchymal transition (EMT) and upregulated MYC in ectopic lesions contribute independently to endometriosis 2015
- LINC01541 Functions as a ceRNA to Modulate the Wnt/β-Catenin Pathway by Decoying miR-506-5p in Endometriosis 2020
- The Evolution of Estrogen Receptor Signaling in the Progression of Endometriosis to Endometriosis-Associated Ovarian Cancer 2018
Cited by (4)
- LINC01541 Functions as a ceRNA to Modulate the Wnt/β-Catenin Pathway by Decoying miR-506-5p in Endometriosis 2020
- The Evolution of Estrogen Receptor Signaling in the Progression of Endometriosis to Endometriosis-Associated Ovarian Cancer 2018
- Endometriosis‐associated infertility: aspects of pathophysiological mechanisms and treatment options 2016
- Enhanced epithelial to mesenchymal transition (EMT) and upregulated MYC in ectopic lesions contribute independently to endometriosis 2015
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- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
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