Endometriose und Adenomyose als Risikofaktoren für ein spontanes Hämatoperitoneum unter der Geburt
This report describes two cases where endometriosis and adenomyosis were risk factors for spontaneous hemoperitoneum during childbirth, requiring immediate delivery, exploratory surgery, and rapid hemostasis to prevent fatal outcomes.
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This case report describes two pregnant patients who experienced spontaneous hemoperitoneum during labor due to ruptured uterine vessels associated with endometriosis and adenomyosis. The first patient, a 43-year-old multipara with both conditions, required an emergency cesarean section after fetal bradycardia, revealing a hemorrhage from a spontaneously ruptured uterine wall vessel. The second patient, a 31-year-old primipara with adenomyosis, developed sudden dizziness and fetal heart rate deceleration during the expulsive phase, leading to the discovery of a hematoperitoneum caused by the rupture of adenomyotic changes in the posterior uterine wall. Both cases highlight that decidualization of ectopic endometrial tissue can cause tissue instability and spontaneous bleeding under shear forces or pressure changes, necessitating immediate delivery and surgical hemostasis to prevent fatal outcomes for mother and child. This paper is centrally about endometriosis and adenomyosis — specifically their role as risk factors for spontaneous hemoperitoneum during childbirth.
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