Editorial: MYC as a disease target beyond cancer
This editorial argues that the transcription factor MYC represents a viable therapeutic target for diseases beyond its well-established role in oncology. The author highlights emerging evidence suggesting that dysregulated MYC activity contributes to various non-cancerous pathological conditions, thereby expanding the scope of potential pharmacological interventions. While acknowledging the historical focus on cancer, the piece advocates for broader investigation into MYC modulation across different disease etiologies. The paper does not explicitly discuss endometriosis or adenomyosis; it was included in the corpus via a keyword match in the upstream search index.
Read from the paper's body, not the abstract. Not a substitute for reading the paper. No clinical advice. How this works
Full text
658 characters
· extracted from
epmc-fulltextxml
· 4 sections
· click to expand
Author
Funding
Conflict
Publisher’S
Text is read by the "Ask this paper" AI Q&A widget below. Extraction quality varies by source — PMC NXML preserves structure cleanly, OA-HTML may include some navigation residue, and OA-PDF can have broken hyphenation. The publisher copy (via DOI) is the canonical version.
My notes (saved in your browser only)
Answers must be backed by verbatim quotes from this paper's full text. Hallucinated quotes are dropped automatically; if no verbatim passage answers the question, we say so. How this works
Citation neighborhood (no data yet)
We don't have any in-corpus citations linked to this paper yet. This is a recent paper (2024) — citers typically take a year or two to land, and the OpenAlex reference graph may still be filling in.
Source provenance
- europepmc
- last seen: 2026-10-04T09:26:46.659050+00:00
- pubmed
- last seen: 2026-10-08T21:45:34.996292+00:00
Per Europe PMC