It Is the Time to Treat Endometriosis Based on Pathophysiology
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AI-generated summary
This paper argues for a shift towards treating endometriosis based on its underlying pathophysiology rather than solely on symptoms.
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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. Outer rings show 2-hop neighbours — papers reached through the immediate citers/citees. [ collapse to 1-hop ]
References (10)
- Apoptosis and bcl-2 expression in normal human endometrium, endometriosis and adenomyosis via openalex
- Endometriosis and the neoplastic process via openalex
- Expression of integrins and E-cadherin in cells from menstrual effluent, endometrium, peritoneal fluid, peritoneum, and endometriosis via openalex
- Genomewide linkage study in 1,176 affected sister pair families identifies a significant susceptibility locus for endometriosis on chromosome 10q26 via openalex
- Metastatic or Embolic Endometriosis, due to the Menstrual Dissemination of Endometrial Tissue into the Venous Circulation via openalex
- Potential involvement of hemoglobin and heme in the pathogenesis of peritoneal endometriosis via openalex
- W2125681453 via openalex
- W2057085814 via openalex
- W2002767054 via openalex
- W2485546735 via openalex
Cited by (1)
Source provenance
- europepmc
- last seen: 2026-10-13T06:10:10.580696+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-10-08T21:24:09.876621+00:00
License: CC0
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