In the Spotlight

Maria Rosa Maduro
In: Reproductive Sciences · 2017 · vol. 24(2) , pp. 173 · doi:10.1177/1933719116682658 · W4234557103
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This paper reviews research priorities and recommendations from international endometriosis research groups and consensus workshops.

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⚙ AI-generated deep summary by claude@2026-06, 2026-06-09 · read from full text ⓘ

The article “In the Spotlight” (Maduro, 2017) is a brief publication that spotlights and contextualizes a set of cited works on endometriosis pathogenesis, clinical burden, research priorities, and experimental or therapeutic approaches. It draws on multiple sources including reviews of disease mechanisms and burden, discussions of surgical management for recurrent symptomatic endometriosis, and preclinical studies targeting pathways such as interleukin-6 signaling, MAPK/VEGFR, and transcriptional regulatory programs. The text provided does not include any explicit study methods, original data, or limitations specific to the “In the Spotlight” piece itself. This paper does not explicitly discuss endometriosis or adenomyosis; it was included in the corpus via a keyword match in the upstream search index.

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Article PDF References Burney R, Giudice L. Pathogenesis and pathophysiology of endo-metriosis. Fertil Steril. 2012;98(3):511–519. Simoens S, Dunselman G, Dirksen C, et al. The burden of endome-triosis: costs and quality of life of women with endometriosis and treated in referral centres. Hum Reprod. 2012;27(5):1292–1299. Vercellini P, Barbara G, Abbiati A, Somigliana E, Vigano P, Fedele L. Repetitive surgery for recurrent symptomatic endometriosis: what to do? Eur J Obstet Gynecol Reprod Biol. 2009;146(1):15–21. Taskin M, Gungor A, Adali E, Yay A, Onder G, Inceboz U. A humanized anti-interleukin 6 receptor monoclonal antibody, toci-lizumab, for the treatment of endometriosis in a rat model. Reprod Sci. 2016;23(5):662–669. Tosti C, Pinzauti S, Santulli P, Chapron C, Petraglia F. Pathoge-netic mechanisms of deep infiltrating endometriosis. Reprod Sci. 2015;22(9):1053–1059. Leconte M, Santulli P, Chouzenoux S, et al. Inhibition of MAPK and VEGFR by sorafenib controls the progression of endometrio-sis. Reprod Sci. 2015;22(9):1171–1180. Yang H, Kang K, Cheng C, Mamillapalli R, Taylor H. Integrative analysis reveals regulatory programs in endometriosis. Reprod Sci. 2015;22(9):1060–1072. Zanatta A, Pereira R, Rocha A, et al. The relationship among HOXA10, estrogen receptor a, progesterone receptor, and progesterone receptor B proteins in rectosigmoid endometriosis: a tissue microarray study. Reprod Sci. 2015;22(1):31–37. Vitagliano A, Noventa M, Quaranta M, Gizzo S. Statins as targeted “magical pills” for the conservative treatment of endome-triosis: may potential adverse effects on female fertility represent the “dark side of the same coin”? A systematic review of literature. Reprod Sci. 2016;23(4):415–428. Rogers P, Adamson G, Al-Jefout M, et al. Research priorities for endometriosis: recommendations from a Global Consortium of Investigators in Endometriosis. Reprod Sci. 2017;24(2):275–299. Rogers P, D’Hooghe T, Fazleabas A, et al. Priorities for endome-triosis research: recommendations from an international consensus workshop. Reprod Sci. 2009;16(4):335–346. Rogers P, D’Hooghe T, Fazleabas A, et al. Defining future directions for endometriosis research: workshop report from the 2011 World Congress of Endometriosis in Montpellier, France. Reprod Sci. 2013;20(5):483–499. Rights and permissions About this article Cite this article Maduro, M.R. In the Spotlight. Reprod. Sci. 24, 173 (2017). https://doi.org/10.1177/1933719116682658 Published: Issue date: DOI: https://doi.org/10.1177/1933719116682658

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Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

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