Transmission and prevention of uterine dysfunction in daughters of dioxin-exposed male mice
Lab / animal
OA: hybrid
public-domain-us
AI-generated summary
A mouse model demonstrates that uterine dysfunction resembling endometriosis can be transmitted from fathers to daughters, potentially via an epigenetic pathway amenable to therapeutic intervention.
One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works
Abstract
In brief: Daughters of women with endometriosis are known to be at increased risk of developing this disease. Using a mouse model, we demonstrate that uterine dysfunction reminiscent of that in endometriosis patients can also be passed from the father to the daughter, potentially via an epigenetic pathway amenable to therapeutic intervention.
My notes (saved in your browser only)
MeSH descriptors
Citation neighborhood (no data yet)
We don't have any in-corpus citations linked to this paper yet. This is a recent paper (2026) — citers typically take a year or two to land, and the OpenAlex reference graph may still be filling in.
SciLite annotations
organisms 2
mus sp.
transgenic mice
Source provenance
- europepmc
- last seen: 2026-10-12T06:17:10.165071+00:00
- openalex
- last seen: 2026-10-12T06:08:43.041439+00:00
- pubmed
- last seen: 2026-10-12T06:10:23.413541+00:00
- scilite
- last seen: 2026-08-30T09:54:48.501902+00:00
License: public-domain-us
· commercial use OK
· attribution required
Courtesy of the U.S. National Library of Medicine
Courtesy of the U.S. National Library of Medicine