Endometriosis
This letter serves as an author reply to a previous publication in the New England Journal of Medicine regarding endometriosis.
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This letter serves as a response to previous correspondence published in the New England Journal of Medicine, addressing specific critiques or comments regarding endometriosis research. The authors defend their prior findings or methodology, engaging in scientific debate within the journal's correspondence section rather than presenting new primary data. The text functions primarily as an academic rebuttal or clarification within the ongoing discourse on the disease. This paper is centrally about endometriosis — specifically serving as a scholarly reply concerning the condition.
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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.
References (9)
- Antiangiogenesis therapy for endometriosis via openalex
- Clinical Activity of Sunitinib and Regorafenib in Endometriosis via openalex
- Effects of Pazopanib, Sunitinib, and Sorafenib, Anti-VEGF Agents, on the Growth of Experimental Endometriosis in Rats via openalex
- Efficacy of Anti-VEGF/VEGFR Agents on Animal Models of Endometriosis: A Systematic Review and Meta-Analysis via openalex
- Reevaluating response and failure of medical treatment of endometriosis: a systematic review via openalex
- W4234160457 via openalex
- W2067050470 via openalex
- W2100086680 via openalex
- W2106932520 via openalex
Cited by (14)
- Suggestive associations between genetically predicted gut microbiota and endometriosis: a two-sample Mendelian randomization study 2026
- Targeting Cathepsin Z suppresses endometriosis via the AMPK/mTOR-mediated autophagy and apoptosis axis 2026
- Comprehensive analysis of tRNA-derived fragment expression in endometriosis using PANDORA-seq technology 2025
- PGK1 mediates glycolysis and cell proliferation in endometriosis by regulating DDIT4 nuclear translocation 2025
- The Interaction of PELP1 With FHL2 Contributes to Ectopic Endometrial Stromal Cell Proliferation, Angiogenesis, and Inflammation in Endometriosis 2025
- Bringing Endometriosis to the Road of Contemporary Pain Science 2025
- Recurrent pain after laparoscopic surgery for ovarian endometrioma: Clinical features and long-term follow up 2024
- Machine Learning-Based Diagnosis And Modeling Research in Endometriosis 2024
- Promotion of BST2 expression by the transcription factor IRF6 affects the progression of endometriosis 2023
- A higher number of exhausted local PD1+, but not TIM3+, NK cells in advanced endometriosis 2023
- Extremely high serum CA19-9 level along with elevated D-dimer in assisting detection of ruptured ovarian endometriosis 2022
- Does Dysmenorrhea Affect Clinical Features and Long-Term Surgical Outcomes of Patients With Ovarian Endometriosis? A 12-Year Retrospective Observational Cohort Study 2022
- Downregulating HK2 inhibits proliferation of endometrial stromal cells through a noncanonical pathway involving phosphorylation of signal transducer and activator of transcription 1 in endometriosis 2022
- Endometriosis and the Urinary Tract: From Diagnosis to Surgical Treatment 2020
Source provenance
- europepmc
- last seen: 2026-11-12T06:12:04.065205+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-10-08T21:27:56.761287+00:00
- unpaywall
- last seen: 2026-11-05T06:08:55.941966+00:00