Getting Endometriosis: A Machinery Ruled by Gene Polymorphism and Epigenetic Mechanisms
This paper explores how gene polymorphism and epigenetic mechanisms contribute to the development of endometriosis.
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This short paper discusses how endometriosis may be driven by an interplay between genetic polymorphisms and epigenetic mechanisms, framing disease susceptibility and tissue behavior in terms of inherited variation and gene regulation changes. It presents a conceptual overview of the “machinery” underlying endometriosis rather than reporting original experimental data, and it does not provide specific study populations or measured outcomes. A stated limitation is that the work is primarily a mechanistic/synthetic discussion, so the evidence is not directly quantified within the paper. This paper is centrally about endometriosis — it focuses on gene polymorphism and epigenetic mechanisms as drivers of endometriosis pathogenesis.
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Cites (4)
- HOX gene expression is altered in the endometrium of women with endometriosis 1999
- The role of the Hoxa10/HOXA10 gene in the etiology of endometriosis and its related infertility: a review 2010
- Endometriosis is associated with aberrant endometrial expression of telomerase and increased telomere length 2008
- The role of survivin in the resistance of endometriotic stromal cells to drug-induced apoptosis 2009
References (5)
- Endometriosis is associated with aberrant endometrial expression of telomerase and increased telomere length via openalex
- HOX gene expression is altered in the endometrium of women with endometriosis via openalex
- The role of survivin in the resistance of endometriotic stromal cells to drug-induced apoptosis via openalex
- The role of the Hoxa10/HOXA10 gene in the etiology of endometriosis and its related infertility: a review via openalex
- W2064751912 via openalex
Source provenance
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00