Role of erythropoietin and its receptor in the development of endometriosis in rats

In: Journal of the Turkish-German Gynecological Association · 2018 · doi:10.4274/jtgga.2018.0039 · W4253924333
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AI-generated summary by gemini-2.5-flash-lite, 2026-06-08

This study investigated the role of erythropoietin and its receptor in the development of endometriosis in a rat model.

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AI-generated deep summary by claude@2026-06, 2026-06-12 · read from full text

The paper investigates the role of erythropoietin and its receptor in the development of endometriosis using a rat model. It focuses on how erythropoietin signaling contributes to endometriosis development, with findings reported to implicate erythropoietin and its receptor in the process. A key limitation is that the work is performed in rats, which may not directly replicate human disease mechanisms. This paper is centrally about endometriosis — it specifically evaluates erythropoietin and its receptor in endometriosis development in rats.

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Abstract

Objective: Besides its hematopoietic function, erythropoietin (EPO) may protect tissues from degenerative disorders. As such, EPO and its receptors were revealed in nonhematopoietic cells, including stromal and endometrial epithelial cells. However, the role of EPO in endometrial disorders is still unknown. Here, we aimed to examine the role of EPO and its receptor activation in the development of endometriosis in rats.
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Condition tags

endometriosis

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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (31)

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last seen: 2026-06-04T00:00:01.174412+00:00
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