Pathological functions of hypoxia in endometriosis
article
OA: closed
CC0
⤵ 8 in-corpus citations
Limited metadata. Only one source feed has indexed
this record so far — no abstract, full text, or open-access copy is
available through Endo Lab. The
publisher's page (linked below)
is the canonical location for the actual content. If you have institutional
access, use "Find at my library".
AI-generated summary
This paper investigates the pathological roles of hypoxia in endometriosis, exploring its contribution to disease development and progression.
One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works
My notes (saved in your browser only)
Condition tags
Citation neighborhood
Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.
Cited by (8)
- HIF-1α regulates DcR3 to promote the development of endometriosis 2024
- The role of iron in the pathogenesis of endometriosis: a systematic review 2023
- The protective effects of Azilsartan against hypoxia in endometrial stromal cells: an implication in endometriosis 2022
- Platelets induce increased estrogen production through NF-κB and TGF-β1 signaling pathways in endometriotic stromal cells 2020
- Endometrial cells contribute to preexisting endometriosis lesions in a mouse model of retrograde menstruation† 2019
- Cyclooxygenase-2 in Endometriosis 2019
- Genomic Function of Estrogen Receptor β in Endometriosis 2019
- Long non‐coding <scp>RNA MALAT</scp>1 mediates hypoxia‐induced pro‐survival autophagy of endometrial stromal cells in endometriosis 2018
Source provenance
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- unpaywall
- last seen: 2026-10-04T06:29:23.997191+00:00
License: CC0
· commercial use OK