Dysmenorrhea and Chronic Pelvic Pain

In: Office Gynecology · 1993 · pp. 135–145 · doi:10.1007/978-1-4612-4340-3_11 · W50251815
book-chapter OA: closed CC0
Full text JSON View on OpenAlex View at publisher
AI-generated summary by claude@2026-06+body, 2026-06-12

Dysmenorrhea, a common gynecologic issue affecting many women, is characterized by painful menstrual flow and is often linked to excessive prostaglandin production causing uterine ischemia.

One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works

AI-generated deep summary by claude@2026-06, 2026-06-12 · read from full text

This chapter reviews dysmenorrhea as the most common gynecologic problem, describing primary (physiologic) dysmenorrhea as painful menses without pelvic disease and noting that excessive endometrial prostaglandin production can contribute to uterine ischemia and hypercontractility. It frames earlier views that treated dysmenorrhea as “psychosomatic” and highlights the use of nonsteroidal anti-inflammatory drugs in effective treatment. A stated limitation is that the discussion is largely conceptual and narrative, with emphasis on general mechanisms and prior work rather than reporting original study methods or outcomes in a specific population. Relevance to endometriosis: the chapter is part of a broader chronic pelvic pain context and cites related gynecologic literature including endometriosis (e.g., diagnosis and pain localization), though the main focus is dysmenorrhea and chronic pelvic pain generally rather than endometriosis itself.

Read from the paper's body, not the abstract. Not a substitute for reading the paper. No clinical advice. How this works

Full text 5,626 characters · extracted from oa-doi-fallback · 3 sections · click to expand

Abstract

Dysmenorrhea, the most common gynecologic problem, means painful monthly flow. It affect between 30% and 75% of all women in the reproductive age group. Primary or physiologic dysmenorrhea is painful menses that occurs in the absence of pelvic disease. It has been shown that excessive production of prostaglandins by the endometrium can result in uterine ischemia and hypercontractibility and can cause severe dysmenorrhea. This has clarified a previous “psychosomatic” disorder, and many strides have been made in effective treatment of dysmenorrhea using nonsteroidal anti-inflammatory medications.1,2 Preview Unable to display preview. Download preview PDF. Similar content being viewed by others

References

Dawood MY. Nonsteroidal anti-inflammatory drugs and changing attitudes toward dysmenorrhea. Am J Med. 1988; 84 (suppl 5A): 23–29. Pasquale SA, Rothhauser R, Dolese HM. A double-blind, placebo controlled study comparing three single-dose regimens of piroxicam with ibuprofen in patients with primary dysmenorrhea. Am J Med. 1988; 84 (suppl 5A): 30–34. Kresch AJ, Seifer DB, Sach LB, et al. Laparoscopy in 100 women with chronic pelvic pain. Obstet Gynecol. 1984; 64: 672. Fedele L, Parazzini F, Bianchi S, et al. Stage and localization of pelvic endometriosis and pain. Fertil Steril. 1990; 53: 155. Melzack R. Neurophysiologic foundations of pain. In: Sternbach RA, ed. The Psychology of Pain. New York: Raven Press; 1986: 1–24. Walker E, Katon W, Harrop-Griffiths H, Holm L, Russ J, Hickok LR. Relationship of chronic pelvic pain to psychiatric diagnoses and childhood sexual abuse. Am J Psychiatry. 1988; 145: 75–80. Reiter RC, Gambone JC. Demographic and historical variables in women with idiopathic chronic pelvic pain. Obstet Gynecol. 1990; 70: 428–432. Reiter RC, Shakerin RL, Gambone JC, Milburn AK. Correlation between sexual abuse and somatization in women with somatic and non-somatic chronic pelvic pain. Am J Obstet Gynecol. 1991; 165: 104–109. Vargyas JM. Chronic pelvic pain. In: Mishell DR, Brenner PF, eds. Management of Common Problems in Obstetrics and Gynecology. NJ: Medical Economics Company Inc; 1988: 322. Steege JF. Assessment and treatment of chronic pelvic pain. In: Thompson JD, Rock JA, eds. TeLindes Operative Gynecology (Update Vol 1, No. 2 ). Philadelphia, Pa: JB Lippincott Company; 1992: 3. Williams TJ, Pratt JH. Endometriosis in 1,000 consecutive celiotomies: incidence and management. Am J Obstet Gynecol. 1977; 116: 245. McArthur JW, Ulfelder H. The effect of pregnancy on endometriosis. Obstet Gynecol Survey. 1965; 20: 709. Nunley WC, Kitchen JD. Endometriosis. In: Sciarra JJ, ed. Obstetrics and Gyencology. Vol 1, no. 20. Philadelphia, Pa: JB Lippincott Company; 1992: 10. Lundorff P, Hahlin M, Kallfelt B, et al. Adhesion formation after laparoscopic surgery in tubal pregnancy: a randomized trial verses laparotomy. Fertil Steril. 1991; 55: 911. Peters AAE, Trimbos-Kemper GLM, Admiral C, Trimbos JB, Hermans J. A randomized clinical trial on the benefit of adhesiolysis patients with intraperitoneal adhesions and chronic pelvic pain. Br J Obstet Gynecol 1991; 99: 59. Andreyko JL, Marshall LA, Kumesic DA, Jaffe RB. Therapeutic uses of gonadotropin releasing hormone analygos. Obstet Gynecol Survey. 1987; 40: 11–12. Beard RW, Highman JH, Pearce S, et al. Diagnosis of pelvic varicosities in women with chronic pelvic pain. Lancet. 1984; 2: 946. Farquhar CM, Rogers V, Franks S, et al. A randomized controlled trial of medroxy-progesterone acetate and psychotherapy for the treatment of pelvic congestion. Br J Obstet Gynaecol. 1989; 96: 1153. Julian TM. Chronic pelvic pain. 1. Workup and diagnosis. The Female Patient. 1989; 14: 37. Edelin KC. Evaluation of female pelvic pain. (pictorial) Hosp Med. 1983 March;19:201–203, 207, 210–212. Precis IV. Washington, DC: American College of Obstetricians and Gynecologists; 1990:15. Anderson F. How Gn Rh agonists facilitate fibroid surgery. Contemp Ob Gyn. 1992: 37: 55–65. TTjaden B, Schlaff WD, Kimball A, Rock JA. The efficacy of presacral neurectomy for the relief of dysmenorrheal. Obstet Gynecol. 1990; 76: 89. Molinak LR. Enometriosis. In: Conn HF, ed. Current Therapy. Philadelphia, Pa: WB Saunders; 1980. Garcia C-r, Davis SS. Pelvic endometriosis: infertility and pelvic pain. Am J Obstet Gynecol. 1997; 129: 740. Lee RB, Stone K, Magelssen D. Presacral neurectomy for chronic pelvic pain. Obstet Gynecol. 1986; 618: 517. Key WR, Hanse LW, Astin M. Argon laser therapy for endometriosis: a review of 92 consecutive patients. Fertil Steril. 1987; 47: 208–212. Steege JF. Assessment and treatment of chronic pelvic pain. In: Thompson JD, Rock JA, eds. TeLindes Operative Gynecology (Update Vol 1, no. 2 ). Philadelphia, Pa: JB Lippincott Company; 1992: 7. Stoval TG, Ling FW, Crawford DA. Hysterectomy for chronic pelvic pain of presumed uterine etiology. Obstet Gynecol. 1990; 75: 676. Editor information Editors and Affiliations Rights and permissions Copyright information © 1993 Springer-Verlag New York, Inc. About this chapter Cite this chapter Saleh, H.J. (1993). Dysmenorrhea and Chronic Pelvic Pain. In: Knaus, J.V., Isaacs, J.H. (eds) Office Gynecology. Springer, New York, NY. https://doi.org/10.1007/978-1-4612-4340-3_11 Download citation DOI: https://doi.org/10.1007/978-1-4612-4340-3_11 Publisher Name: Springer, New York, NY Print ISBN: 978-1-4612-8740-7 Online ISBN: 978-1-4612-4340-3 eBook Packages: Springer Book Archive

Keywords

These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

Text is read by the "Ask this paper" AI Q&A widget below. Extraction quality varies by source — PMC NXML preserves structure cleanly, OA-HTML may include some navigation residue, and OA-PDF can have broken hyphenation. The publisher copy (via DOI) is the canonical version.

My notes (saved in your browser only)

Ask this paper AI returns verbatim quotes from the full text · source: oa-doi-fallback

Answers must be backed by verbatim quotes from this paper's full text. Hallucinated quotes are dropped automatically; if no verbatim passage answers the question, we say so. How this works

Condition tags

chronic_pelvic_paindysmenorrhea

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (27)

Source provenance

openalex
last seen: 2026-06-10T17:14:06.276822+00:00
License: CC0 · commercial use OK