Salpingitis isthmica nodosa und Adenomyosis tubae
Salpingitis isthmica nodosa, a thickening of the fallopian tube's isthmus, is usually inflammatory, while adenomyosis tubae involves uterine-derived adenomyosis or heterotopic endometrium without inflammation.
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The paper describes knot-like thickening at the isthmic end of the fallopian tube, arguing that in most cases it originates from inflammation, with deep epithelial invasion, tubule formation, and muscle hyperplasia as consequences, sometimes with abscess formation, and that the term “salpingitis isthmica nodosa” is therefore appropriate. In rare cases, the thickening has a different origin: it can represent uterine adenomyosis spreading into the tube, or heterotopic endometrial tissue within the tubal lumen producing glandular invagination into the tubal musculature, leading the author to use the term “adenomyosis tubae.” The paper notes that even adenomyosis tubae does not exclude later onset of inflammation and that such rare non-inflammatory cases would not be suitable for studies aimed at elucidating the causes of tubal swelling. Relevance to endometriosis: while primarily focused on tubal pathology and adenomyosis spreading to the tube, it explicitly addresses adenomyosis tubae and thus relates directly to adenomyosis.
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- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
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