Recurrent Scar Endometriosis Developing in the episiotomy

Esengul Türkyılmaz, Gökhan S. Kılıç, Fatma Doğa Öcal, Seda Maralı, Ayşe Filiz Yavuz Avşar
In: Medeniyet Medical Journal · 2017 · doi:10.5222/mmj.2017.1004 · W2604467305
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⚙ AI-generated summary by claude@2026-06+body, 2026-06-08 ⓘ

This case report details a patient with recurrent endometriosis in an episiotomy scar, found to be deeply embedded and consistent with endometriosis upon pathological examination.

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⚙ AI-generated deep summary by claude@2026-06, 2026-06-09 · read from full text ⓘ

This paper reports a case of recurrent scar endometriosis that developed in an episiotomy scar one year after surgical treatment for the same condition. The patient presented with cyclic pain and swelling localized to a mass embedded about 2.5 cm deep in the prior episiotomy scar, which was excised circumferentially, and pathology confirmed endometriosis in fibro-adipose tissues. The authors describe continuity of endometriotic cells as possibly driven by increased steroidogenic acute regulatory protein production via prostaglandin E2 receptor signaling and higher aromatase levels, leading to local hyperestrogenism, and they note genetic predisposition as a contributor to recurrence. This paper is centrally about endometriosis — specifically recurrent endometriosis developing in an episiotomy scar with cyclic symptoms and proposed mechanisms of persistence/recurrence.

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Abstract The present case reported a patient having with recurrent endometriotic lesion which occurred in the episiotomy scar. The patient who was operated due to episiotomy scar endometriosis one year ago applied to our outpatient clinic with cyclic pain and swelling especially during menstrual periods which started on the same area. The mass ingrained approximately 2.5 cm deep from the surface, which localized on the episiotomy scar, was reached and circumferentially excised. In the pathological examination, tissue which embedded into the fibro-adipose tissues which was consistent with endometriosis, was detected. Increased steroidogenic acute regulatory protein production by Prostoglandin E2 receptor and the level of aromatase provide continuity of the endometriotic cells which were implanted during the vaginal birth with local hiperestrogenism. The genetic predisposition probably increases the tendency of recurrence of endometriotic lesions. This two mechanisms may be considered responsible for current case.

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Condition tags

endometriosis

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (11)

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last seen: 2026-06-04T00:00:01.174412+00:00
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