Peter Sutovsky

No ORCID on file · 4 papers in corpus · active 2008-2022

Study types

  • article 2
  • other 2

Condition tags

  • endometriosis 4
  • infertility 1
article 2022
A re-appraisal of mesenchymal-epithelial transition (MET) in endometrial epithelial remodeling
Cell and tissue research ·doi:10.1007/s00441-022-03711-z

Mesenchymal-epithelial transition (MET) is a mechanism of endometrial epithelial regeneration. It is also implicated in adenocarcinoma and endometriosis. Little is known about this process in normal uterine physiology. Previously, using pre…

other 2012
Cell biology solves mysteries of reproduction
Cell and tissue research ·doi:10.1007/s00441-012-1480-y

Reproduction and fertility have been objects of keen inquiry since the dawn of humanity. Medieval anatomists provided the first accurate depictions of the female reproductive system, and early microscopists were fascinated by the magnified …

other 2010
Neutralizing TIMP1 restores fecundity in a rat model of endometriosis and treating control rats with TIMP1 causes anomalies in ovarian function and embryo development
Biology of reproduction ·doi:10.1095/biolreprod.109.083287

Human and rat endometriotic lesions synthesize and secrete tissue inhibitor of metalloproteinase 1 (TIMP1). More TIMP1 localizes in the ovarian theca in an established rat model for endometriosis (Endo) when compared to surgical controls (S…

article 2008
Reduced Fecundity in Female Rats with Surgically Induced Endometriosis and in Their Daughters: A Potential Role for Tissue Inhibitors of Metalloproteinase 11
Biology of reproduction ·doi:10.1095/biolreprod.108.073411

The cause of reduced fecundity in women with endometriosis is unknown. Expression of matrix metalloproteinases (MMPs) and tissue inhibitors of metalloproteinases (TIMPs) by both ectopic and eutopic endometrium reportedly has a role in the p…