Qiong Wu

No ORCID on file · 8 papers in corpus · active 2022-2026

Study types

  • article 3
  • other 2
  • erratum 1
  • preprint 1

Condition tags

  • endometriosis 7
  • chronic_pelvic_pain 1
  • die_deep_infiltrating 1
other 2026
CX3CR1/P2X4R signaling-mediated spinal microglial M1 polarization contributes to chronic pelvic pain in endometriosis
International immunopharmacology ·doi:10.1016/j.intimp.2026.116471

BACKGROUND: Deeply infiltrating endometriosis (DIE), characterized by its extrauterine invasion, is strongly associated with chronic pelvic pain (CPP). Peripheral-central sensitization synergistically contribute to the pathogenesis of CPP i…

other 2026
EGR1 promotes ferroptosis in endometriosis through transcriptional activation of HMOX1
Molecular and cellular endocrinology ·doi:10.1016/j.mce.2025.112721

Endometriosis (EM) affects approximately 10% of women of reproductive age and remains a prevalent estrogen-dependent gynecological disorder with limited therapeutic efficacy and high recurrence rates. Ferroptosis-an iron-dependent, non-apop…

erratum 2025
Correction: Macrophages originated IL-33/ST2 inhibits ferroptosis in endometriosis via the ATF3/SLC7A11 axis
Cell death & disease ·doi:10.1038/s41419-025-07484-5
2025
The context-dependent epigenetic and organogenesis programs determine 3D vs. 2D cellular fitness of MYC-driven murine liver cancer cells
·doi:10.21203/rs.3.rs-4390765/v4

Abstract 3D cellular-specific epigenetic and transcriptomic reprogramming is critical to organogenesis and tumorigenesis. Here we dissect the distinct cell fitness in 2D (normoxia vs. chronic hypoxia) vs 3D (normoxia) culture conditions …

article 2024
IGF2BP3 promotes glutamine metabolism of endometriosis by interacting with UCA1 to enhances the mRNA stability of GLS1
Molecular medicine (Cambridge, Mass.) ·doi:10.1186/s10020-024-00834-7

BACKGROUND: Insulin like growth factor II mRNA binding protein 3 (IGF2BP3) has been implicated in numerous inflammatory and cancerous conditions. However, its precise molecular mechanisms in endometriosis (EMs) remains unclear. The aim of t…

article 2023
Macrophages originated IL-33/ST2 inhibits ferroptosis in endometriosis via the ATF3/SLC7A11 axis
Cell death & disease ·doi:10.1038/s41419-023-06182-4

Endometriosis is a gynecological inflammatory disease that is linked with immune cells, specifically macrophages. IL-33 secreted from macrophages is known to accelerate the progression of endometriosis. The periodic and repeated bleeding th…

preprint 2023
Macrophages originated IL33/ST2 inhibits ferroptosis in endometriosis via the ATF3/SLC7A11 axis
·doi:10.21203/rs.3.rs-2835730/v1

Abstract Endometriosis is a gynecological inflammatory disease which linked with immune cells, specifically macrophages. And IL-33 secreted from macrophages is known to accelerate the progression of endometriosis. The periodic and repeated …

article 2022
Silencing of lncRNA MALAT1 facilitates erastin-induced ferroptosis in endometriosis through miR-145-5p/MUC1 signaling
Cell death discovery ·doi:10.1038/s41420-022-00975-w

Endometriosis is a chronic disorder characterized by the implantation of endometrial glands and stroma outside the uterus. However, the pathogenesis of endometriosis is still unclear. To date, there is no fully effective treatment without t…