Song-Juan Dai

No ORCID on file · 7 papers in corpus · active 2017-2024

Study types

  • other 4
  • dataset 2
  • article 1

Condition tags

  • endometriosis 7
other 2024
Immunology ·doi:10.1111/imm.13786

Endometriosis is defined as an oestrogen-dependent and inflammatory gynaecological disease of which the pathogenesis remains unclear. This study aimed to investigate the cellular heterogeneity and reveal the effect of CD8+ T cells on the pr…

other 2017
Journal of Obstetrics and Gynaecology Research ·doi:10.1111/jog.13201

AIM: Lipoxin A4 (LXA4 ) can function as an endogenous 'breaking signal' in inflammation and plays an important role in the progression of endometriosis. The proteome responses to interleukin-1β (IL-1β) or LXA4 in human endometriotic stromal…

article 2017
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1177/1933719117718271
other 2017
·doi:10.25384/sage.c.4106783

Objective:Epithelial–mesenchymal transition (EMT) is essential for embryogenesis, fibrosis, and tumor metastasis. Aberrant EMT phenomenon has been reported in endometriotic tissues of patients with endometriosis (EM). In this study, we furt…

other 2017
·doi:10.25384/sage.c.4106783.v1

Objective:Epithelial–mesenchymal transition (EMT) is essential for embryogenesis, fibrosis, and tumor metastasis. Aberrant EMT phenomenon has been reported in endometriotic tissues of patients with endometriosis (EM). In this study, we furt…

dataset 2017
·doi:10.25384/sage.6294656

Supplementary Material for Lipoxin A4 Suppresses Estrogen-Induced Epithelial–Mesenchymal Transition via ALXR-Dependent Manner in Endometriosis by Rong-Feng Wu, Zhi-Xiong Huang, Jing Ran, Song-Juan Dai, Dian-Chao Lin, Tai-Wei Ng, Qing-Xi Che…

dataset 2017
·doi:10.25384/sage.6294656.v1

Supplementary Material for Lipoxin A4 Suppresses Estrogen-Induced Epithelial–Mesenchymal Transition via ALXR-Dependent Manner in Endometriosis by Rong-Feng Wu, Zhi-Xiong Huang, Jing Ran, Song-Juan Dai, Dian-Chao Lin, Tai-Wei Ng, Qing-Xi Che…