Su E

No ORCID on file · 4 papers in corpus
article 2014
ERβ- and Prostaglandin E2-Regulated Pathways Integrate Cell Proliferation via Ras-like and Estrogen-Regulated Growth Inhibitor in Endometriosis
Molecular Endocrinology ·doi:10.1210/me.2013-1421

In endometriosis, stromal and epithelial cells from the endometrium form extrauterine lesions and persist in response to estrogen (E2) and prostaglandin E2 (PGE2). Stromal cells produce excessive quantities of estrogen and PGE2 in a feed-fo…

other 2012
Activated glucocorticoid and eicosanoid pathways in endometriosis
Fertility and sterility ·doi:10.1016/j.fertnstert.2012.03.030

OBJECTIVE: To define altered gene expression networks in endometriosis. DESIGN: Experiments using endometriotic tissues and primary cells. SETTING: Division of Reproductive Biology Research, Northwestern University. PATIENT(S): Premenopa…

review 2008
Steroidogenic factor-1 and endometriosis
Molecular and cellular endocrinology ·doi:10.1016/j.mce.2008.12.012

Endometriosis is a common and chronic disease characterized by persistent pelvic pain and infertility. Estradiol is essential for growth and inflammation in endometriotic tissue. The complete cascade of steroidogenic proteins/enzymes includ…

2007
Aromatase excess in cancers of breast, endometrium and ovary.
The Journal of steroid biochemistry and molecular biology ·doi:10.1016/j.jsbmb.2007.05.027

Pathogenesis and growth of three common women's cancers (breast, endometrium and ovary) are linked to estrogen. A single gene encodes the key enzyme for estrogen biosynthesis named aromatase, inhibition of which effectively eliminates estro…