New perspectives about the article “Dual inhibition of AKT and ERK1/2 pathways restores the expression of progesterone Receptor-B in endometriotic lesions through epigenetic mechanisms”
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This paper explores dual inhibition of AKT and ERK1/2 pathways and its role in restoring progesterone receptor-B expression in endometriotic lesions via epigenetic mechanisms.
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References (8)
- Estetrol Increases Progesterone Genetic Response without Triggering Common Estrogenic Effects in Endometriotic Cell Lines and Primary Cultures via openalex
- Estetrol Inhibits Endometriosis Development in an In Vivo Murine Model via openalex
- Notch-1 Signaling Activation and Progesterone Receptor Expression in Ectopic Lesions of Women With Endometriosis via openalex
- Progesterone resistance in endometriosis: A pathophysiological perspective and potential treatment alternatives via openalex
- Surgical versus low-dose progestin treatment for endometriosis-associated severe deep dyspareunia II: Effect on sexual functioning, psychological status and health-related quality of life via openalex
- W4366332545 via openalex
- W4399221424 via openalex
- W2083063093 via openalex
Source provenance
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
License: CC0
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