Additional file 4 of IGF2BP3 promotes glutamine metabolism of endometriosis by interacting with UCA1 to enhances the mRNA stability of GLS1
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IGF2BP3 promotes glutamine metabolism in endometriosis by interacting with UCA1, enhancing the mRNA stability of GLS1.
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Supplementary Material 4.
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References (52)
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- Estrogen receptor β upregulates CCL2 via NF-κB signaling in endometriotic stromal cells and recruits macrophages to promote the pathogenesis of endometriosis via openalex
- Metabolic Profile of Patients with Severe Endometriosis: a Prospective Experimental Study via openalex
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- Rethinking mechanisms, diagnosis and management of endometriosis via openalex
- Silencing of lncRNA MALAT1 facilitates erastin-induced ferroptosis in endometriosis through miR-145-5p/MUC1 signaling via openalex
- The burden of endometriosis: costs and quality of life of women with endometriosis and treated in referral centres via openalex
- Transforming Growth Factor-β Induced Warburg-Like Metabolic Reprogramming May Underpin the Development of Peritoneal Endometriosis via openalex
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- last seen: 2026-05-13T20:12:55.465734+00:00
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