Correction: CHIP induces ubiquitination and degradation of HMGB1 to regulate glycolysis in ovarian endometriosis
This correction identifies the co-corresponding authors for a paper investigating how CHIP induces HMGB1 ubiquitination and degradation to regulate glycolysis in ovarian endometriosis.
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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.
Cited by (5)
- PRMT3-mediated FOXO1 arginine methylation exacerbates oxidative stress-induced decidualization defects in the eutopic endometrium of endometriosis 2025
- Ubiquitination of PFKFB4 by CHIP regulates glycolysis and progression in endometriosis† 2025
- PRMT3-mediated FOXO1 arginine methylation exacerbates oxidative stress-induced decidualization defects in the eutopic endometrium of endometriosis 2025
- Ubiquitination of PFKFB4 by CHIP regulates glycolysis and progression in endometriosis† 2025
- Ubiquitination of PFKFB4 by CHIP Regulates Glycolysis and Progression in Endometriosis 2024
Source provenance
- europepmc
- last seen: 2026-08-03T06:10:56.557307+00:00
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- pubmed
- last seen: 2026-08-03T06:10:17.514410+00:00