Dienogest enhances autophagy induction in endometriotic cells by impairing activation of AKT, ERK1/2, and MTOR
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Dienogest promotes autophagy in endometriotic cells by inhibiting the activation of AKT, ERK1/2, and MTOR signaling pathways.
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Cited by (7)
- The Multifactorial Pathogenesis of Endometriosis: A Narrative Review Integrating Hormonal, Immune, and Microbiome Aspects 2025
- How transcription factors regulate apoptosis in endometriosis (Review) 2025
- The "Road" to Malignant Transformation from Endometriosis to Endometriosis-Associated Ovarian Cancers (EAOCs): An mTOR-Centred Review 2024
- SCM-198 Prevents Endometriosis By Reversing Low Autophagy Levels Of Endometriotic Stromal Cells Via Inhibiting TNF-Α-Aromatase-Estrogen-Erα Pathway And Promoting PR Expression 2022
- Role of Endometrial Autophagy in Physiological and Pathophysiological Processes 2019
- Expression Profile Analysis of Circular RNAs in Ovarian Endometriosis by Microarray and Bioinformatics 2018
- Long non-coding RNA MALAT1 mediates hypoxia-induced pro-survival autophagy of endometrial stromal cells in endometriosis 2018
Source provenance
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00
- unpaywall
- last seen: 2026-10-31T06:28:23.984441+00:00
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