Correction: Bone marrow mesenchymal stem cells combined with estrogen synergistically promote endometrial regeneration and reverse EMT via Wnt/β-catenin signaling pathway
This correction updates Figs. 3 and 4 of the original article, which found that bone marrow mesenchymal stem cells combined with estrogen synergistically promote endometrial regeneration by reversing EMT via the Wnt/β-catenin signaling pathway.
One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works
This correction notice updates an earlier Reproductive Biology and Endocrinology article by Yuan et al., in which an error was identified in Figs. 3 and 4; the corrected figures have been provided and the original article updated. The underlying study evaluated bone marrow mesenchymal stem cell transplantation time and assessed fibrosis-related protein markers (including N-cadherin, Collagen I, TGF-β1, and α-SMA by western blot) across time points, along with histological assessment of endometrial gland morphology and intrauterine fibrosis after injury using HE and Masson staining. A stated limitation is that the correction reflects corrected figure content (and includes an explanation for cropping of gels/blots in Fig. 3), rather than a full re-analysis of all results. Relevance to endometriosis: the paper’s original title and content focus on estrogen-assisted endometrial regeneration and EMT reversal through Wnt/β-catenin signaling, which is directly related to endometrial pathophysiology implicated in endometriosis.
Read from the paper's body, not the abstract. Not a substitute for reading the paper. No clinical advice. How this works
My notes (saved in your browser only)
Citation neighborhood (no data yet)
We don't have any in-corpus citations linked to this paper yet. The paper's references may be in our DB but unresolved to ``paper_id`` (resolution happens at ingest when the cited DOI matches a row we already have). Run the cross-source citation reconcile pass to retry.
References (1)
- W4291511918 via openalex
Source provenance
- openalex
- last seen: 2026-06-10T17:14:06.276822+00:00