Endometriosis Intestinal
This article outlines the unresolved etiology of intestinal endometriosis, presenting two primary theoretical frameworks proposed by various authors. Sampson’s theory suggests that endometrial cells reflux through the uterine tubes and subsequently implant on the peritoneum or adjacent pelvic viscera. Alternatively, Ivanoff and Meyer propose a metaplasia hypothesis where peritoneal serosal cells transform into endometrial cells due to their shared embryonic origin. This paper is centrally about endometriosis — specifically focusing on the theoretical mechanisms underlying intestinal involvement.
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