Preperitoneal Fat Thicknesses, Lipid Profile, and Oxidative Status in Women With Uterine Fibroids

In: Reproductive Sciences · 2017 · vol. 24(10) , pp. 1419–1425 · doi:10.1177/1933719116689598 · PMID:28122481 · W2581740772
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AI-generated summary by claude@2026-06+body, 2026-06-11

Women with uterine fibroids showed increased preperitoneal fat thickness, altered lipid profiles, higher oxidative stress, and lower antioxidant activity compared to controls.

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This cross-sectional study evaluated 35 women with uterine fibroids and 15 controls, measuring anthropometrics and ultrasonographic preperitoneal fat thickness (PFT) and subcutaneous fat thickness, alongside plasma lipid profile (including HDL-C, LDL-C, and oxidized LDL), glucose, and oxidative stress markers (TBARs) and antioxidative status (SOD activity). Women with uterine fibroids had higher PFT, lower HDL-C, higher LDL-C and oxidized LDL, higher TBARs, and lower SOD activity, and PFT was positively associated with BMI, oxidized LDL, and TBARs; in multivariate analysis, PFT and HDL-C remained correlated with fibroid diagnosis. The paper’s main caveat is that its cross-sectional design limits causal inference regarding whether visceral fat–related inflammation drives fibroid development. Relevance to endometriosis/adenomyosis: the paper primarily focuses on uterine fibroids, but it is included in this endometriosis/adenomyosis research corpus because it discusses oxidative stress and inflammation, mechanisms also implicated in endometriosis and adenomyosis in related literature.

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Abstract

Purpose There is growing evidence supporting a possible role for metabolic syndrome and its determinants, such as dyslipidemia, in uterine fibroid (UF) pathogenesis. The present study aims to investigate the association between UFs and visceral and subcutaneous fat thickness (SFT), lipid profile, and oxidative and antioxidative status.

Methods

In this cross-sectional study, 35 patients diagnosed with UFs and 15 women without UFs were enrolled. Clinical history and anthropometric parameters were collected for every woman. Characteristics of UFs, preperitoneal fat thickness (PFT), and SFT were assessed ultrasonically. Lipid profile, glucose, thiobarbituric acid reactive substances (TBARs), and superoxide dismutase (SOD) activity were evaluated on plasma from participants.

Results

Women with UFs showed a significantly increased PFT (11.63 ± 3.39 vs 7.01 ± 3.10 mm; P <.001), lower levels of high-density lipoprotein cholesterol (HDL-C; 45.4 ± 8.3 vs 57.2 ± 13.4 mg/dL; P =.017), higher levels of low-density lipoprotein cholesterol (LDL-C; 92.3 ± 21.5 vs 72.0 ± 14.6 mg/dL; P =.007), and oxidized LDL (65.2 ± 20.7 vs 43.0 ± 11.3 U/L; P =.002). In patients, TBARs concentration was significantly higher (9.41 ± 6.49 vs 2.92 ± 1.65 nmol malondialdehyde/100 μg prot; P <.001), whereas SOD activity was lower (1.09 ± 0.19 vs 1.37 ± 0.41 U/μL; P =.005). Preperitoneal fat thickness was positively associated with body mass index, oxidized LDL, and TBARs. At multivariate analysis, PFT and HDL-C maintained a significant correlation with the diagnosis of UFs.

Conclusion

Chronic inflammation triggered and sustained by visceral fat could play a determinant role in cell differentiation and proliferation processes, necessary for the development of UFs. Alterations in cholesterol fractions may be explained as a consequence of the increased visceral fat deposits and can reflect an increased risk of subclinical atherosclerosis in patients with UF. Similar content being viewed by others

References

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