Progesterone-Mediated Reduction of Endometriotic Lesions in Experimental Model of Endometriosis Can be Disrupted by Dioxin (TCDD) Via Interleukin 1α Pathway

K Osteen
In: Fertility and Sterility · 1997 · vol. 68 , pp. S12 · doi:10.1016/s0015-0282(97)84739-5 · W4231577943
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In an experimental endometriosis model, dioxin (TCDD) disrupts the progesterone-mediated reduction of endometriotic lesions by activating the interleukin 1α pathway.

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