Developmental dioxin exposure promotes a hyper-inflammatory peritoneal microenvironment which may contribute to the endometriosis-like phenotype

Dana R. Glore; Kaylon L. Bruner‐Tran; K. Boyd; Jacqueline Lucas; Kevin G. Osteen

doi:10.1016/j.fertnstert.2012.07.119

Developmental dioxin exposure promotes a hyper-inflammatory peritoneal microenvironment which may contribute to the endometriosis-like phenotype

Dana R. Glore, Kaylon L. Bruner‐Tran, K. Boyd, Jacqueline Lucas, Kevin G. Osteen

In: Fertility and Sterility · 2012 · vol. 98(3) , pp. S32 · doi:10.1016/j.fertnstert.2012.07.119 · W2064673370

article OA: bronze CC0 ⤵ 1 in-corpus citation

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⚙ AI-generated summary by claude@2026-06, 2026-06-10 ⓘ

Developmental exposure to dioxins increased peritoneal inflammation and immune cell infiltration, potentially contributing to endometriosis-like symptoms.

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endometriosis

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Cited by (1)

Exposure to the environmental endocrine disruptor TCDD and human reproductive dysfunction: Translating lessons from murine models 2016

Cited by (1)

Exposure to the environmental endocrine disruptor TCDD and human reproductive dysfunction: Translating lessons from murine models 2016

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