EXPRESSION PROFILING AND FUNCTIONAL ANALYSIS REVEAL THAT CELL CYCLE PROGRESSION PATHWAYS ARE HIGHLY UPREGULATED IN MURINE ENDOMETRIOSIS LESIONS DEFICIENT IN CD55

Elliott G. Richards; Emily Esakov; Jenna M. Rehmer; Ofer Reizes

doi:10.1016/j.fertnstert.2021.07.302

EXPRESSION PROFILING AND FUNCTIONAL ANALYSIS REVEAL THAT CELL CYCLE PROGRESSION PATHWAYS ARE HIGHLY UPREGULATED IN MURINE ENDOMETRIOSIS LESIONS DEFICIENT IN CD55

Elliott G. Richards, Emily Esakov, Jenna M. Rehmer, Ofer Reizes

In: Fertility and Sterility · 2021 · vol. 116(3) , pp. e108–e109 · doi:10.1016/j.fertnstert.2021.07.302 · W3200855191

OA: closed CC0 ⤵ 1 in-corpus citation

🔓 Open OA copy View on OpenAlex View at publisher

⚙ AI-generated summary by qwen3.7-flash, 2026-08-21 ⓘ

This study analyzed murine endometriosis lesions deficient in CD55, revealing that cell cycle progression pathways are highly upregulated through expression profiling and functional analysis.

One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works

My notes (saved in your browser only)

Condition tags

endometriosis

Citation neighborhood (sparse)

Too few in-corpus citations on either side for a chart; here are the lists.

Cited by (1)

Potential shared pathogenic mechanisms between endometriosis and inflammatory bowel disease indicate a strong initial effect of immune factors 2024

Cited by (1)

Potential shared pathogenic mechanisms between endometriosis and inflammatory bowel disease indicate a strong initial effect of immune factors 2024

Source provenance

openalex: last seen: 2026-06-10T17:14:06.276822+00:00
unpaywall: last seen: 2026-09-17T06:32:46.214484+00:00

License: CC0 · commercial use OK