Rapid regression of endometriosis by a new gonadotropin-releasing hormone antagonist in rats with surgically induced disease.

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Cited by (7)

• Identifying Mechanisms of Endometriosis-Associated Reduced Fecundity in a Rat Model: Novel Insights toward Understanding Human Infertility 2020
• Elagolix, a Novel, Orally Bioavailable GnRH Antagonist under Investigation for the Treatment of Endometriosis-Related Pain 2015
• Developmental exposure of fetal ovaries and fetal germ cells to endometriosis in an endometriosis model causes differential gene expression in the preimplantation embryos of the first-generation and second-generation embryos 2013
• Using Rats as a Research Model for the Study of Endometriosis 2002
• Gonadotropin-Releasing Hormone Agonist (GnRH-a) Therapy Alters Activity of Plasminogen Activators, Matrix Metalloproteinases, and Their Inhibitors in Rat Models for Adhesion Formation and Endometriosis: Potential GnRH-a–Regulated Mechanisms Reducing Adhesion Formation 1998
• Spontaneous and steroid-induced recurrence of endometriosis after suppression by a gonadotropin-releasing hormone antagonist in the rat 1991
• Follicular Atresia and Infertility in Rats Treated with a Gonadotropin-Releasing Hormone Antagonist* 1990

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