Differential effects of interferon-α and -β on the secretion of vascular endothelial growth factor by eutopic and ectopic endometrial stromal cells
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Interferon-alpha and -beta differentially regulate vascular endothelial growth factor secretion from eutopic and ectopic endometrial stromal cells.
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Abstract
Interferon (IFN)-beta, not IFN-alpha, suppresses vascular endothelial growth factor secretion in endometriotic cyst stromal cells. It was suggested that IFN-beta may be more suitable than IFN-alpha as an anti-endometriotic agent.
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Too few in-corpus citations on either side for a chart; here are the lists.
Cites (4)
- Antiangiogenic Agents Are Effective Inhibitors of Endometriosis 2003
- Pathogenetic Significance of Increased Levels of Interleukin-8 in the Peritoneal Fluid of Patients with Endometriosis 1998
- Serum concentrations of growth factors in women with and without endometriosis: the action of anti-endometriosis medicines 2003
- Use of intraperitoneal interferon α-2b therapy after conservative surgery for endometriosis and postoperative medical treatment with depot gonadotropin-releasing hormone analog: a randomized clinical trial 2002
Cited by (3)
- Endometriotic cells are resistant to interferon-γ-induced cell growth inhibition and apoptosis: a possible mechanism involved in the pathogenesis of endometriosis 2004
- Endometriosis: harmful survival of an ectopic tissue 2006
- Interferons: pathogenetic rationale for the treatment of external genital endometriosis and clinical efficacy 2019
References (7)
- Antiangiogenic Agents Are Effective Inhibitors of Endometriosis via openalex
- Pathogenetic Significance of Increased Levels of Interleukin-8 in the Peritoneal Fluid of Patients with Endometriosis via openalex
- Serum concentrations of growth factors in women with and without endometriosis: the action of anti-endometriosis medicines via openalex
- Use of intraperitoneal interferon α-2b therapy after conservative surgery for endometriosis and postoperative medical treatment with depot gonadotropin-releasing hormone analog: a randomized clinical trial via openalex
- W2042214171 via openalex
- W2128523336 via openalex
- W2135769202 via openalex
Cited by (3)
- Interferons: pathogenetic rationale for the treatment of external genital endometriosis and clinical efficacy 2019
- Endometriosis: harmful survival of an ectopic tissue 2006
- Endometriotic cells are resistant to interferon-γ-induced cell growth inhibition and apoptosis: a possible mechanism involved in the pathogenesis of endometriosis 2004
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- openalex
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- last seen: 2026-06-02T02:00:03.124865+00:00
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