Parthenolide reduces cell proliferation and prostaglandin E₂ synthesis in human endometriotic stromal cells and inhibits development of endometriosis in the murine model
Parthenolide was found to reduce cell proliferation and prostaglandin E2 synthesis in human endometriotic stromal cells and inhibit endometriosis development in a murine model.
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This study investigated the effects of parthenolide on human endometriotic stromal cells and a murine model of endometriosis. The researchers found that parthenolide significantly reduced cell proliferation and decreased the synthesis of prostaglandin E2 in the cultured human cells. In vivo experiments using mice demonstrated that treatment with parthenolide inhibited the development of endometriotic lesions. This paper is centrally about endometriosis — specifically, the pharmacological inhibition of lesion development and cellular proliferation by parthenolide.
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- last seen: 2026-06-04T00:00:01.174412+00:00